Periodontal Gum Treatment disease in smokers can be difficult. The immune system is compromised, and there is less blood flow to the gums, limiting the ability of infection-fighting white blood cells to reach the area. It also delays the healing of teeth and gum tissue. While it is not known exactly how smoking affects the periodontal microflora, it has been observed that smokers tend to have a higher rate of periodontal pathogens.
Tobacco use is one of the leading risk factors for developing gum disease. This type of oral disease is caused by bacteria. Smoking has been linked to several serious diseases including heart disease and oral cancer. Fortunately, tobacco use has been decreasing over the past few decades.
One of the best ways to decrease your risk of gum disease is to quit smoking. But, quitting is not easy. Nicotine, the main component of cigarettes, has a high addiction potential, so it is hard to kick the habit. Instead, you may want to seek other periodontal gum treatment options. Taking antibiotics and treating the condition in its early stages can help prevent tooth loss and bone deterioration around the teeth. In addition, a healthy diet rich in vitamin C is important.
The presence of Fusobacterium, an intermediate colonizer, is often detected in the subgingival microflora in smokers. These bacteria may bridge the attachment of commensals with pathogenic bacteria and may promote the formation of biofilms. A study suggests that they may transport periodontal pathogens to the oral cavity and enhance their growth.
Smoking is also thought to decrease the number of blood vessels in the gums. Consequently, less blood flow means that there is not enough blood to support the healing of gum tissue. This can lead to gum recession and a more agresive periodontal gum treatment.
There have been many studies on the effects of smoking on the immune system, and many of them have shown that tobacco use impairs the host’s ability to fight infection. Researchers also found that smokers have less of the defensive blood cells, such as antibodies and T-cells, which help the body fight infections. However, more research is needed to determine the exact underlying mechanisms with a periodontal gum treatment.
There are a number of common medical conditions that increase the risk of developing periodontitis. Some of the more common risk factors include older age, diabetes, malnutrition, and autoimmune disorders. Additionally, smokers are more likely to develop a more severe form of gum disease than non-smokers.
The effect of tobacco smoke on the oral cavity has been studied by a variety of researchers. Researchers found that nicotine inhibited the inflammatory response of human umbilical vein endothelial cells. And, high concentrations of nicotine were associated with lower gingival bleeding indices in smoking periodontitis patients. Another study showed that nicotine tipped the balance between MMPs and TIMPs, which are key mediators of inflammation.
Smoking has also been shown to affect the microbial function of pathogen-host cell interactions. Researchers have found that nicotine interferes with the ability of the cells to respond to the infection, and this leads to more damage to the tissue.
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